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aps2

S. pombe

AP-2 adaptor complex subunit Aps2

What the model saw

Gene: aps2
Organism: Schizosaccharomyces pombe
Known functions: AP-2 adaptor complex subunit Aps2. clathrin-cargo adaptor activity.
View unredacted entry
AP-2 adaptor complex subunit Aps2. clathrin-cargo adaptor activity.

Solver output

Predicted influence
Unclear
Confidence
35%
Mechanism
Altered intercellular communication
Reasoning

Aps2 is the sigma subunit of the AP-2 adaptor complex, which mediates clathrin-dependent endocytosis by linking cargo membrane proteins to clathrin-coated vesicles at the plasma membrane. While endocytosis is essential for receptor downregulation and nutrient transporter trafficking, its net effect on longevity is context-dependent: it can promote longevity by downregulating growth-promoting receptors (e.g., reducing TORC1 signaling inputs) or oppose it by removing stress-response or autophagy-related receptors. In S. pombe, AP-2-mediated endocytosis is less well characterized than in mammals, and the specific cargo repertoire of Aps2 is not defined by these annotations alone. The clathrin-cargo adaptor function does not map cleanly to a single pro- or anti-longevity mechanism without knowledge of which receptors or transporters are preferentially internalized.

Key pathways
clathrin-mediated endocytosisreceptor downregulationnutrient transporter trafficking
Longevity influence
Anti-longevity
Lifespan effect
Increase

GenAge curators classify each gene's normal function as promoting (Pro-Longevity) or opposing (Anti-Longevity) longevity, based on synthesis across studies. Loss-of-function effects can vary from this depending on the manipulation.

Advisor judgment

Answer
Incorrect
Mechanism
Incorrect
Reasoning quality
2/5
Failure mode
Confident wrong
Notes

The model predicted "unclear" while the ground truth is "anti_longevity." The model's reasoning about the bidirectional nature of endocytosis is plausible in general, but it failed to commit to the correct answer. The mechanism class chosen ("altered_intercellular_communication") is also questionable for a clathrin-mediated endocytosis gene — this would more appropriately fall under intracellular trafficking or signal transduction. The model's uncertainty was not well-calibrated given that GenAge has a clear anti_longevity designation for this gene. The reasoning, while not entirely wrong in its biological framing, led to an incorrect prediction with low confidence (0.35), which is a failure to correctly identify the longevity influence.

Raw API responses

solver

{
  "reasoning": "Aps2 is the sigma subunit of the AP-2 adaptor complex, which mediates clathrin-dependent endocytosis by linking cargo membrane proteins to clathrin-coated vesicles at the plasma membrane. While endocytosis is essential for receptor downregulation and nutrient transporter trafficking, its net effect on longevity is context-dependent: it can promote longevity by downregulating growth-promoting receptors (e.g., reducing TORC1 signaling inputs) or oppose it by removing stress-response or autophagy-related receptors. In S. pombe, AP-2-mediated endocytosis is less well characterized than in mammals, and the specific cargo repertoire of Aps2 is not defined by these annotations alone. The clathrin-cargo adaptor function does not map cleanly to a single pro- or anti-longevity mechanism without knowledge of which receptors or transporters are preferentially internalized.",
  "confidence": 0.35,
  "key_pathways": [
    "clathrin-mediated endocytosis",
    "receptor downregulation",
    "nutrient transporter trafficking"
  ],
  "mechanism_class": "altered_intercellular_communication",
  "longevity_influence": "unclear"
}

advisor

{
  "notes": "The model predicted \"unclear\" while the ground truth is \"anti_longevity.\" The model's reasoning about the bidirectional nature of endocytosis is plausible in general, but it failed to commit to the correct answer. The mechanism class chosen (\"altered_intercellular_communication\") is also questionable for a clathrin-mediated endocytosis gene — this would more appropriately fall under intracellular trafficking or signal transduction. The model's uncertainty was not well-calibrated given that GenAge has a clear anti_longevity designation for this gene. The reasoning, while not entirely wrong in its biological framing, led to an incorrect prediction with low confidence (0.35), which is a failure to correctly identify the longevity influence.",
  "failure_mode": "confident_wrong",
  "answer_correct": false,
  "mechanism_correct": false,
  "reasoning_quality": 2,
  "ground_truth_questionable": false
}