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cbp-3

C. elegans

histone acetyltransferase

What the model saw

Gene: cbp-3
Organism: Caenorhabditis elegans
Known functions: histone acetyltransferase. histone acetyltransferase activity. protein-lysine-acetyltransferase activity.
View unredacted entry
histone acetyltransferase. histone acetyltransferase activity. protein-lysine-acetyltransferase activity.

Solver output

Predicted influence
Pro-longevity
Confidence
65%
Mechanism
Epigenetic alterations
Reasoning

cbp-3 encodes a histone acetyltransferase (HAT) in C. elegans, orthologous to the CBP/p300 family of transcriptional coactivators. HATs like CBP/p300 acetylate histone lysine residues (e.g., H3K27ac, H3K18ac), opening chromatin and enabling transcription of stress-response and longevity-associated genes. In C. elegans, CBP-1 (the primary CBP ortholog) is known to cooperate with DAF-16/FOXO to promote longevity downstream of the insulin/IGF-1 signaling pathway; cbp-3, as a related HAT, likely participates in similar epigenetic activation of pro-longevity transcriptional programs. Maintenance of appropriate histone acetylation patterns is critical to counteract age-associated epigenetic drift and heterochromatin loss. Loss of HAT activity in aging organisms is associated with transcriptional silencing of stress-response genes, supporting a pro-longevity role for cbp-3's normal function.

Key pathways
histone acetylation / chromatin remodelingDAF-16/FOXO transcriptional activationinsulin/IGF-1 longevity signaling
Longevity influence
Pro-longevity
Lifespan effect
Decrease

GenAge curators classify each gene's normal function as promoting (Pro-Longevity) or opposing (Anti-Longevity) longevity, based on synthesis across studies. Loss-of-function effects can vary from this depending on the manipulation.

Advisor judgment

Answer
Correct
Mechanism
Correct
Reasoning quality
3/5
Failure mode
Correct
Notes

The model correctly predicted pro_longevity for cbp-3. The mechanistic reasoning is plausible and grounded in known C. elegans biology (CBP/p300 family HATs, DAF-16/FOXO cooperation, epigenetic regulation of longevity). However, the reasoning is somewhat speculative - the model explicitly notes cbp-3 is a "related" HAT to CBP-1 and uses hedged language ("likely participates"), suggesting it's extrapolating from CBP-1 rather than citing direct evidence for cbp-3 specifically. The confidence of 0.65 is appropriately modest given this uncertainty. Overall a reasonable prediction with sound mechanistic logic, though not strongly evidence-based for cbp-3 specifically.

Raw API responses

solver

{
  "reasoning": "cbp-3 encodes a histone acetyltransferase (HAT) in C. elegans, orthologous to the CBP/p300 family of transcriptional coactivators. HATs like CBP/p300 acetylate histone lysine residues (e.g., H3K27ac, H3K18ac), opening chromatin and enabling transcription of stress-response and longevity-associated genes. In C. elegans, CBP-1 (the primary CBP ortholog) is known to cooperate with DAF-16/FOXO to promote longevity downstream of the insulin/IGF-1 signaling pathway; cbp-3, as a related HAT, likely participates in similar epigenetic activation of pro-longevity transcriptional programs. Maintenance of appropriate histone acetylation patterns is critical to counteract age-associated epigenetic drift and heterochromatin loss. Loss of HAT activity in aging organisms is associated with transcriptional silencing of stress-response genes, supporting a pro-longevity role for cbp-3's normal function.",
  "confidence": 0.65,
  "key_pathways": [
    "histone acetylation / chromatin remodeling",
    "DAF-16/FOXO transcriptional activation",
    "insulin/IGF-1 longevity signaling"
  ],
  "mechanism_class": "epigenetic_alterations",
  "longevity_influence": "pro_longevity"
}

advisor

{
  "notes": "The model correctly predicted pro_longevity for cbp-3. The mechanistic reasoning is plausible and grounded in known C. elegans biology (CBP/p300 family HATs, DAF-16/FOXO cooperation, epigenetic regulation of longevity). However, the reasoning is somewhat speculative - the model explicitly notes cbp-3 is a \"related\" HAT to CBP-1 and uses hedged language (\"likely participates\"), suggesting it's extrapolating from CBP-1 rather than citing direct evidence for cbp-3 specifically. The confidence of 0.65 is appropriately modest given this uncertainty. Overall a reasonable prediction with sound mechanistic logic, though not strongly evidence-based for cbp-3 specifically.",
  "failure_mode": "correct",
  "answer_correct": true,
  "mechanism_correct": true,
  "reasoning_quality": 3,
  "ground_truth_questionable": false
}
cbp-3 · Caenorhabditis elegans | Aging biology eval